Please use this identifier to cite or link to this item: https://ahro.austin.org.au/austinjspui/handle/1/10512
Title: Mechanisms and clinical features of internal watershed infarction.
Austin Authors: Baird, A E;Donnan, Geoffrey A ;Saling, Michael M 
Affiliation: Department of Neurology, Austin Hospital, Heidelberg, Victoria.
Issue Date: 16-May-1991
Publication information: Clinical and Experimental Neurology; 28(): 50-5
Abstract: The mechanism of internal carotid watershed cerebral infarction is not well understood, but the phenomenon has been described in association with carotid occlusive disease, and more recently with distal middle cerebral artery occlusion beyond the origin of the lenticulostriate branches. The clinical correlates of these changes have not yet been described. We present 5 patients in whom acute internal watershed infarction had occurred, and correlate the clinical, neuropsychological and 99mTc-HMPAO SPECT (Single Photon Emission Computed Tomography using 99mtechnetium-hexamethylpropylene amine oxime) cerebral perfusion findings. Four patients had distal middle cerebral artery occlusion demonstrated on angiography, and one showed profound hemispheric depression in cerebral perfusion with only a small area of infarction. We have concentrated on the mechanism of distal middle cerebral artery occlusion to describe the "arc" of the watershed zone created. We propose that internal watershed infarcts can further be subdivided into anterior and posterior subtypes, outline the vascular territories involved, and propose an overall classification of cerebral watershed infarction.
Gov't Doc #: 1821839
URI: http://ahro.austin.org.au/austinjspui/handle/1/10512
URL: https://pubmed.ncbi.nlm.nih.gov/1821839
Type: Journal Article
Subjects: Adult
Aged
Cerebral Infarction.physiopathology
Cerebrovascular Circulation
Female
Humans
Male
Middle Aged
Tomography, Emission-Computed, Single-Photon
Tomography, X-Ray Computed
Appears in Collections:Journal articles

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