Please use this identifier to cite or link to this item: https://ahro.austin.org.au/austinjspui/handle/1/10512
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dc.contributor.authorBaird, A Een
dc.contributor.authorDonnan, Geoffrey Aen
dc.contributor.authorSaling, Michael Men
dc.date.accessioned2015-05-15T23:58:47Z
dc.date.available2015-05-15T23:58:47Z
dc.date.issued1991-05-16en
dc.identifier.citationClinical and Experimental Neurology; 28(): 50-5en
dc.identifier.govdoc1821839en
dc.identifier.otherPUBMEDen
dc.identifier.urihttp://ahro.austin.org.au/austinjspui/handle/1/10512en
dc.description.abstractThe mechanism of internal carotid watershed cerebral infarction is not well understood, but the phenomenon has been described in association with carotid occlusive disease, and more recently with distal middle cerebral artery occlusion beyond the origin of the lenticulostriate branches. The clinical correlates of these changes have not yet been described. We present 5 patients in whom acute internal watershed infarction had occurred, and correlate the clinical, neuropsychological and 99mTc-HMPAO SPECT (Single Photon Emission Computed Tomography using 99mtechnetium-hexamethylpropylene amine oxime) cerebral perfusion findings. Four patients had distal middle cerebral artery occlusion demonstrated on angiography, and one showed profound hemispheric depression in cerebral perfusion with only a small area of infarction. We have concentrated on the mechanism of distal middle cerebral artery occlusion to describe the "arc" of the watershed zone created. We propose that internal watershed infarcts can further be subdivided into anterior and posterior subtypes, outline the vascular territories involved, and propose an overall classification of cerebral watershed infarction.en
dc.language.isoenen
dc.subject.otherAdulten
dc.subject.otherAgeden
dc.subject.otherCerebral Infarction.physiopathologyen
dc.subject.otherCerebrovascular Circulationen
dc.subject.otherFemaleen
dc.subject.otherHumansen
dc.subject.otherMaleen
dc.subject.otherMiddle Ageden
dc.subject.otherTomography, Emission-Computed, Single-Photonen
dc.subject.otherTomography, X-Ray Computeden
dc.titleMechanisms and clinical features of internal watershed infarction.en
dc.typeJournal Articleen
dc.identifier.journaltitleClinical and experimental neurologyen
dc.identifier.affiliationDepartment of Neurology, Austin Hospital, Heidelberg, Victoria.en
dc.description.pages50-5en
dc.relation.urlhttps://pubmed.ncbi.nlm.nih.gov/1821839en
dc.type.austinJournal Articleen
item.fulltextNo Fulltext-
item.openairetypeJournal Article-
item.languageiso639-1en-
item.cerifentitytypePublications-
item.grantfulltextnone-
item.openairecristypehttp://purl.org/coar/resource_type/c_18cf-
crisitem.author.deptThe Florey Institute of Neuroscience and Mental Health-
crisitem.author.deptClinical Neuropsychology-
crisitem.author.deptThe Florey Institute of Neuroscience and Mental Health-
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