Please use this identifier to cite or link to this item: https://ahro.austin.org.au/austinjspui/handle/1/31779
Title: Plasma p-tau181/Aβ1-42 ratio predicts Aβ-PET status and correlates with CSF-p-tau181/Aβ1-42 and future cognitive decline.
Austin Authors: Fowler, Christopher J;Stoops, Erik;Rainey-Smith, Stephanie R;Vanmechelen, Eugeen;Vanbrabant, Jeroen;Dewit, Nele;Mauroo, Kimberley;Maruff, Paul;Rowe, Christopher C ;Fripp, Jurgen;Li, Qiao-Xin;Bourgeat, Pierrick;Collins, Steven J;Martins, Ralph N;Masters, Colin L ;Doecke, James D
Affiliation: The Florey Institute of Neuroscience and Mental Health
ADx NeuroSciences Ghent Belgium.
School of Medical and Health Sciences Centre of Excellence for Alzheimer's Disease Research & Care Edith Cowan University Joondalup Western Australia Australia.
Cogstate Ltd Melbourne Victoria Australia.
Australian E-Health Research Centre CSIRO Herston Queensland Australia.
Department of Medicine (RMH) The University of Melbourne Melbourne Victoria Australia.
Issue Date: 2022
Date: 2022
Publication information: Alzheimer's and Dementia 2022
Abstract: In Alzheimer's disease (AD), plasma amyloid beta (Aβ)1-42 and phosphorylated tau (p-tau) predict high amyloid status from Aβ positron emission tomography (PET); however, the extent to which combination of these plasma assays can predict remains unknown.
URI: https://ahro.austin.org.au/austinjspui/handle/1/31779
DOI: 10.1002/dad2.12375
ORCID: 0000-0003-2863-0293
Journal: Alzheimer's and Dementia
Start page: e12375
PubMed URL: 36447478
Type: Journal Article
Subjects: Alzheimer's disease
amyloid beta amyloid imaging
blood biomarkers
blood diagnostic for Alzheimer's disease
cerebrospinal fluid
phosphorylated tau
plasma amyloid beta
plasma phosphorylated tau181
positron emission tomography
Appears in Collections:Journal articles

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