Please use this identifier to cite or link to this item: https://ahro.austin.org.au/austinjspui/handle/1/33660
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dc.contributor.authorWarren, Josephine-
dc.contributor.authorDinh, Diem-
dc.contributor.authorBrennan, Angela-
dc.contributor.authorTan, Christianne-
dc.contributor.authorDagan, Misha-
dc.contributor.authorStehli, Julia-
dc.contributor.authorClark, David J-
dc.contributor.authorAjani, Andrew E-
dc.contributor.authorReid, Christopher M-
dc.contributor.authorSebastian, Martin-
dc.contributor.authorOqueli, Ernesto-
dc.contributor.authorFreeman, Melanie-
dc.contributor.authorStub, Dion-
dc.contributor.authorDuffy, Stephen J-
dc.date2023-
dc.date.accessioned2023-09-06T07:03:28Z-
dc.date.available2023-09-06T07:03:28Z-
dc.date.issued2023-11-
dc.identifier.citationHypertension (Dallas, Tex. : 1979) 2023-11; 80(11)en_US
dc.identifier.issn1524-4563-
dc.identifier.urihttps://ahro.austin.org.au/austinjspui/handle/1/33660-
dc.description.abstractRecent US guidelines recommend lower blood pressure (BP) targets in hypertension, but aggressive lowering of diastolic BP (DBP) can occur at the expense of myocardial perfusion, particularly in the presence of coronary artery disease. We sought to establish the long-term impact of low DBP on mortality among patients undergoing percutaneous coronary intervention with well-controlled systolic BP. We analyzed data from 12 965 patients undergoing percutaneous coronary intervention between 2009 and 2018 from the Melbourne Interventional Group registry who had a preprocedural systolic BP of ≤140 mm Hg. Patients with ST-elevation myocardial infarction, cardiogenic shock, and out-of-hospital arrest were excluded. Patients were stratified into 5 groups according to preprocedural DBP: <50, 50 to 59, 60 to 69, 70 to 79, and ≥80 mm Hg. The primary outcome was long-term, all-cause mortality. Mortality data were derived from the Australian National Death Index. Patients with DBP<50 mm Hg were older with higher rates of diabetes, renal impairment, prior myocardial infarction, left ventricular dysfunction, peripheral and cerebrovascular disease (all P<0.001). Patients with DBP<50 mm Hg had higher 30-day (2.5% versus 0.7% for the other 4 quintiles; P<0.0001) and long-term mortality (median, 3.6 years; follow-up, 29% versus 11%; P<0.0001). Cox-regression analysis revealed that DBP<50 mm Hg was an independent predictor of long-term mortality (HR, 1.55 [95% CI, 1.20-2.00]; P=0.001). In patients with well-controlled systolic BP undergoing percutaneous coronary intervention, low DBP (<50 mm Hg) is an independent predictor of long-term mortality.en_US
dc.language.isoeng-
dc.subjectcoronary artery diseaseen_US
dc.subjectdiastolic blood pressureen_US
dc.subjectlong-term outcomesen_US
dc.subjectperfusionen_US
dc.subjectregistriesen_US
dc.titleImpact of Preprocedural Diastolic Blood Pressure on Outcomes in Patients Undergoing Percutaneous Coronary Intervention.en_US
dc.typeJournal Articleen_US
dc.identifier.journaltitleHypertension (Dallas, Tex. : 1979)en_US
dc.identifier.affiliationDepartment of Cardiology, Alfred Hospital, Melbourne, Australia (J.W., C.T., M.D., J.S., D.S.).en_US
dc.identifier.affiliationCentre of Cardiovascular Research and Education in Therapeutics, Monash University, Melbourne, Australia (D.D., A.B., C.M.R., D.S., S.J.D.).en_US
dc.identifier.affiliationCardiologyen_US
dc.identifier.affiliationDepartment of Cardiology, Royal Melbourne Hospital, Melbourne, Australia (A.E.A.).en_US
dc.identifier.affiliationDepartment of Cardiology, Barwon Health, Geelong, Australia (M.S.).en_US
dc.identifier.affiliationDepartment of Cardiology, Grampians Health Ballarat, Australia (E.O.).;School of Medicine, Faculty of Health, Deakin University, Geelong, VIC, Australia (E.O.).en_US
dc.identifier.affiliationDepartment of Cardiology, Box Hill Hospital, Melbourne, Australia (M.F.).en_US
dc.identifier.affiliationCentre of Cardiovascular Research and Education in Therapeutics, Monash University, Melbourne, Australia (D.D., A.B., C.M.R., D.S., S.J.D.).en_US
dc.identifier.doi10.1161/HYPERTENSIONAHA.123.20963en_US
dc.type.contentTexten_US
dc.identifier.orcid0000-0003-4788-2277en_US
dc.identifier.orcid0000-0002-8167-3906en_US
dc.identifier.orcid0000-0003-1375-7187en_US
dc.identifier.orcid0000-0002-2353-9776en_US
dc.identifier.orcid0000-0003-3089-1062en_US
dc.identifier.orcid0000-0001-9173-3944en_US
dc.identifier.orcid0000-0001-8686-2709en_US
dc.identifier.orcid0000-0002-4518-5948en_US
dc.identifier.pubmedid37655489-
item.cerifentitytypePublications-
item.openairetypeJournal Article-
item.languageiso639-1en-
item.fulltextNo Fulltext-
item.openairecristypehttp://purl.org/coar/resource_type/c_18cf-
item.grantfulltextnone-
crisitem.author.deptCardiology-
crisitem.author.deptUniversity of Melbourne Clinical School-
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