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Please use this identifier to cite or link to this item: https://ahro.austin.org.au/austinjspui/handle/1/28913
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dc.contributor.authorIsles, Nicole S-
dc.contributor.authorMu, Andre-
dc.contributor.authorKwong, Jason C-
dc.contributor.authorHowden, Benjamin P-
dc.contributor.authorStinear, Timothy P-
dc.date2022-
dc.date.accessioned2022-03-01T04:43:54Z-
dc.date.available2022-03-01T04:43:54Z-
dc.date.issued2022-02-18-
dc.identifier.citationTrends in Microbiology 2022; 30(9): 853-865en
dc.identifier.urihttps://ahro.austin.org.au/austinjspui/handle/1/28913-
dc.description.abstractThe human gut is host to a diverse range of microorganisms that offer protection against colonization by multidrug-resistant bacteria. Antibiotic use, medications, health conditions, and lifestyle factors can alter the composition of the gut microbiota in such a way that results in loss of colonization resistance and increased susceptibility to invading pathogenic antibiotic-resistant bacteria. Therapeutics aiming to restore a diverse and protective microbiome are fast advancing. In this review, we focus on the compositional changes within the gut microbiome that are associated with colonization resistance and discuss their use as potential targets for therapeutics or diagnostics.en
dc.language.isoeng-
dc.subjectcolonization resistanceen
dc.subjectmicrobiomeen
dc.subjectmultidrug-resistant organism (MDRO)en
dc.titleGut microbiome signatures and host colonization with multidrug-resistant bacteria.en
dc.typeJournal Articleen
dc.identifier.journaltitleTrends in microbiologyen
dc.identifier.affiliationMicrobiological Diagnostic Unit Public Health Laboratory, The University of Melbourne at the Peter Doherty Institute for Infection and Immunity, Victoria, 3000, Australia..en
dc.identifier.affiliationMolecular and Systems Physiology Laboratory, The Salk Institute for Biological Studies, La Jolla, CA 92037, USA..en
dc.identifier.affiliationDepartment of Microbiology and Immunology, The University of Melbourne at the Peter Doherty Institute for Infection and Immunity, Victoria, 3000, Australia..en
dc.identifier.affiliationDoherty Applied Microbial Genomics, The University of Melbourne at the Peter Doherty Institute for Infection and Immunity, Victoria, 3000, Australia..en
dc.identifier.affiliationDepartment of Medicine, The University of Melbourne, Melbourne, Australia..en
dc.identifier.affiliationInfectious Diseasesen
dc.identifier.affiliationGene Expression Laboratory, The Salk Institute for Biological Studies, La Jolla, CA 92037, USA..en
dc.identifier.affiliationNOMIS Center for Immunobiology and Microbial Pathogenesis, The Salk Institute for Biological Studies, La Jolla, CA 92037, USA..en
dc.identifier.pubmedurihttps://pubmed.ncbi.nlm.nih.gov/35184972/en
dc.identifier.doi10.1016/j.tim.2022.01.013en
dc.type.contentTexten
dc.identifier.orcid0000-0002-6298-7942en
dc.identifier.orcid0000-0003-0237-1473en
dc.identifier.pubmedid35184972-
local.name.researcherHowden, Benjamin P
item.fulltextNo Fulltext-
item.grantfulltextnone-
item.openairetypeJournal Article-
item.languageiso639-1en-
item.openairecristypehttp://purl.org/coar/resource_type/c_18cf-
item.cerifentitytypePublications-
crisitem.author.deptInfectious Diseases-
crisitem.author.deptInfectious Diseases-
crisitem.author.deptMicrobiology-
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