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Please use this identifier to cite or link to this item: https://ahro.austin.org.au/austinjspui/handle/1/16644
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dc.contributor.authorManning, Todd G-
dc.contributor.authorCheung, Ernest-
dc.contributor.authorPerera, Marlon-
dc.contributor.authorChristidis, Daniel-
dc.contributor.authorO'Brien, Jonathan S-
dc.contributor.authorMitchell, Catherine-
dc.contributor.authorBolton, Damien M-
dc.contributor.authorLawrentschuk, Nathan-
dc.date2017-04-21-
dc.date.accessioned2017-05-04T01:56:51Z-
dc.date.available2017-05-04T01:56:51Z-
dc.date.issued2017-09-
dc.identifier.citationUrology 2017; 107: 5-10en_US
dc.identifier.urihttps://ahro.austin.org.au/austinjspui/handle/1/16644-
dc.description.abstractMultiparametric Magnetic Resonance Imaging (mpMRI) has added to the armamentarium for the diagnosis and surveillance for organ confined prostate cancer. Atypical small acinar proliferation (ASAP) and high-grade prostatic intraepithelial neoplasia (HGPIN) are premalignant prostatic lesions. The management of such lesions remains contentious, and the addition of mpMRI introduces further uncertainty given its ability to pick up indolent lesions and its use in targeted biopsy. We aimed to perform a comprehensive review of current evidence regarding ASAP, HGPIN and mpMRI to ascertain a consensus for a current management algorithm.en_US
dc.subjectASAPen_US
dc.subjectAtypical small acinar proliferationen_US
dc.subjectHGPINen_US
dc.subjectHigh-grade prostatic intraepithelial neoplasiaen_US
dc.subjectMultiparametric Magnetic Resonance Imagingen_US
dc.subjectPremalignant lesionsen_US
dc.titleAtypical small acinar proliferation and high grade prostatic intraepithelial neoplasia in the era of multi-parametric MRI: a contemporary reviewen_US
dc.typeJournal Article-
dc.identifier.journaltitleUrologyen_US
dc.identifier.affiliationDepartment of Surgery, Austin Health, The University of Melbourne, Heidelberg, Victoria, Australiaen
dc.identifier.affiliationThe Young Urology Researchers Organisation (YURO), Melbourne, Victoria, Australiaen
dc.identifier.affiliationDepartment of Pathology, Peter MacCallum Cancer Centre, Melbourne, Victoria, Australiaen
dc.identifier.affiliationDepartment of Surgical Oncology, Peter MacCallum Cancer Centre, Melbourne, Victoria, Australiaen
dc.identifier.affiliationOlivia Newton-John Cancer Research Institute, Heidelberg, Victoria, Australiaen
dc.identifier.pubmedurihttps://pubmed.ncbi.nlm.nih.gov/28438627en_US
dc.identifier.doi10.1016/j.urology.2017.04.021en_US
dc.type.contentTexten_US
dc.identifier.orcid0000-0002-1138-6389en
dc.identifier.orcid0000-0003-2951-3726en
dc.identifier.orcid0000-0001-5609-3769en
dc.identifier.orcid0000-0002-5145-6783en
dc.identifier.orcid0000-0001-8553-5618en
dc.type.austinJournal Articleen_US
dc.type.austinReview-
local.name.researcherBolton, Damien M
item.cerifentitytypePublications-
item.openairecristypehttp://purl.org/coar/resource_type/c_18cf-
item.fulltextNo Fulltext-
item.openairetypeJournal Article-
item.grantfulltextnone-
crisitem.author.deptUrology-
crisitem.author.deptRadiology-
crisitem.author.deptSurgery-
crisitem.author.deptUrology-
crisitem.author.deptUrology-
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