Please use this identifier to cite or link to this item: https://ahro.austin.org.au/austinjspui/handle/1/13410
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dc.contributor.authorGilbert, Richard Een
dc.contributor.authorCox, Allison Jen
dc.contributor.authorDziadek, Men
dc.contributor.authorCooper, Mark Een
dc.contributor.authorJerums, Georgeen
dc.date.accessioned2015-05-16T03:15:21Z
dc.date.available2015-05-16T03:15:21Z
dc.date.issued1995-10-12en
dc.identifier.citationJournal of Diabetes and Its Complications; 9(4): 252-4en
dc.identifier.govdoc8573740en
dc.identifier.otherPUBMEDen
dc.identifier.urihttp://ahro.austin.org.au/austinjspui/handle/1/13410en
dc.description.abstractIncreased extracellular matrix (ECM) is the ultrastructural hallmark of diabetic microangiopathy. Its accumulation within the kidney is directly linked to the clinical manifestations of diabetic nephropathy, namely proteinuria and declining renal function. The pathogenesis of ECM changes in diabetes is not well understood, but is likely to involve interaction between cells, growth factors, structural proteins, and cell receptors for these molecules. Molecular biological techniques may offer the necessary tools for gaining insight into the pathogenetic processes that eventually lead to renal failure in diabetes.en
dc.language.isoenen
dc.subject.otherAnimalsen
dc.subject.otherCell Adhesionen
dc.subject.otherDiabetic Nephropathies.physiopathologyen
dc.subject.otherExtracellular Matrix.physiologyen
dc.subject.otherExtracellular Matrix Proteins.physiologyen
dc.subject.otherGrowth Substances.physiologyen
dc.subject.otherHumansen
dc.subject.otherKidney.physiopathologyen
dc.subject.otherProteinuriaen
dc.titleExtracellular matrix and its interactions in the diabetic kidney: a molecular biological approach.en
dc.typeJournal Articleen
dc.identifier.journaltitleJournal of diabetes and its complicationsen
dc.identifier.affiliationEndocrinology Unit Austin Hospital, Victoria, Australiaen
dc.description.pages252-4en
dc.relation.urlhttps://pubmed.ncbi.nlm.nih.gov/8573740en
dc.type.austinJournal Articleen
item.languageiso639-1en-
item.openairecristypehttp://purl.org/coar/resource_type/c_18cf-
item.openairetypeJournal Article-
item.grantfulltextnone-
item.cerifentitytypePublications-
item.fulltextNo Fulltext-
crisitem.author.deptEndocrinology-
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