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Please use this identifier to cite or link to this item: https://ahro.austin.org.au/austinjspui/handle/1/12357
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dc.contributor.authorMcIntyre, I Men
dc.contributor.authorOxenkrug, G Fen
dc.date.accessioned2015-05-16T02:02:48Z
dc.date.available2015-05-16T02:02:48Z
dc.date.issued1989-11-01en
dc.identifier.citationPharmacopsychiatry; 22(6): 263-5en
dc.identifier.govdoc2515551en
dc.identifier.otherPUBMEDen
dc.identifier.urihttps://ahro.austin.org.au/austinjspui/handle/1/12357en
dc.description.abstractThe effects of the antidepressant compound bupropion were studied on rat pineal indoleamines. A pharmacologically relevant dose of bupropion either acutely (20 mg/kg) or chronically (20 mg/kg/day for 12 days) had no significant effect on pineal melatonin synthesis or other pineal indoleamine concentrations. The significance of this finding is discussed in relation to the lack of effect of bupropion on beta-adrenergic receptors.en
dc.language.isoenen
dc.subject.otherAnimalsen
dc.subject.otherAntidepressive Agents.pharmacologyen
dc.subject.otherBiogenic Monoamines.metabolismen
dc.subject.otherBupropionen
dc.subject.otherIndoles.metabolismen
dc.subject.otherMaleen
dc.subject.otherMelatonin.metabolismen
dc.subject.otherPineal Gland.drug effects.metabolismen
dc.subject.otherPropiophenones.pharmacologyen
dc.subject.otherRatsen
dc.subject.otherRats, Inbred Strainsen
dc.titleLack of effect of the antidepressant compound bupropion on pineal indoleamines.en
dc.typeJournal Articleen
dc.identifier.journaltitlePharmacopsychiatryen
dc.identifier.affiliationDepartment of Psychiatry, University of Melbourne & Austin Hospital, Heidelberg, Australiaen
dc.description.pages263-5en
dc.relation.urlhttps://pubmed.ncbi.nlm.nih.gov/2515551en
dc.type.austinJournal Articleen
item.openairetypeJournal Article-
item.cerifentitytypePublications-
item.openairecristypehttp://purl.org/coar/resource_type/c_18cf-
item.fulltextNo Fulltext-
item.grantfulltextnone-
item.languageiso639-1en-
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