Please use this identifier to cite or link to this item: https://ahro.austin.org.au/austinjspui/handle/1/10741
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dc.contributor.authorBadawy, Radwa A Ben
dc.contributor.authorHarvey, A Simonen
dc.contributor.authorMacdonell, Richard A Len
dc.date.accessioned2015-05-16T00:17:14Z-
dc.date.available2015-05-16T00:17:14Z-
dc.date.issued2009-01-04en
dc.identifier.citationJournal of Clinical Neuroscience 2009; 16(3): 355-65en
dc.identifier.govdoc19124246en
dc.identifier.otherPUBMEDen
dc.identifier.urihttps://ahro.austin.org.au/austinjspui/handle/1/10741en
dc.description.abstractEpilepsy encompasses a diverse group of seizure disorders caused by a variety of structural, cellular and molecular alterations of the brain primarily affecting the cerebral cortex, leading to recurrent unprovoked epileptic seizures. In this two-part review we examine the mechanisms underlying normal neuronal function and those predisposing to recurrent epileptic seizures starting at the most basic cellular derangements (Part 1) and working up to the highly complex epileptic networks (Part 2). We attempt to show that multiple factors can modify the epileptic process and that different mechanisms underlie different types of epilepsy, and in most situations there is an interplay between multiple genetic and environmental factors.en
dc.language.isoenen
dc.subject.otherAnimalsen
dc.subject.otherCerebral Cortex.pathology.physiopathologyen
dc.subject.otherEpilepsy.etiology.pathologyen
dc.subject.otherHumansen
dc.subject.otherIon Channel Gating.physiologyen
dc.subject.otherIon Channels.classification.physiologyen
dc.subject.otherNeuroglia.physiologyen
dc.subject.otherNeurons.physiologyen
dc.subject.otherSynaptic Transmission.physiologyen
dc.titleCortical hyperexcitability and epileptogenesis: understanding the mechanisms of epilepsy - part 1.en
dc.typeJournal Articleen
dc.identifier.journaltitleJournal of Clinical Neuroscienceen
dc.identifier.affiliationDepartment of Neurology, Austin Health, Heidelberg, Victoria, Australiaen
dc.identifier.doi10.1016/j.jocn.2008.08.026en
dc.description.pages355-65en
dc.relation.urlhttps://pubmed.ncbi.nlm.nih.gov/19124246en
dc.type.austinJournal Articleen
local.name.researcherMacdonell, Richard A L
item.languageiso639-1en-
item.fulltextNo Fulltext-
item.grantfulltextnone-
item.openairecristypehttp://purl.org/coar/resource_type/c_18cf-
item.cerifentitytypePublications-
item.openairetypeJournal Article-
crisitem.author.deptNeurology-
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