Please use this identifier to cite or link to this item: https://ahro.austin.org.au/austinjspui/handle/1/12840
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dc.contributor.authorHowes, L Gen
dc.contributor.authorHodsman, G Pen
dc.date.accessioned2015-05-16T02:35:11Z
dc.date.available2015-05-16T02:35:11Z
dc.date.issued1989-08-01en
dc.identifier.citationClinical and Experimental Pharmacology & Physiology; 16(8): 671-4en
dc.identifier.govdoc2791336en
dc.identifier.otherPUBMEDen
dc.identifier.urihttp://ahro.austin.org.au/austinjspui/handle/1/12840en
dc.description.abstract1. The effects of low or high sodium intake for 4 weeks on cardiac catecholamines and 3,4-dihydroxyphenylethylene glycol (DHPG) levels were studied in adult female Wistar rats. 2. Rats receiving the low sodium diet had significantly higher plasma renin activity than rats receiving the high sodium diet. 3. Dopamine concentrations in both the right and left ventricle were significantly higher in the low salt compared with the high salt rats, but noradrenaline, adrenaline and DHPG levels did not differ significantly between the two groups. 4. These data do not support previous reports that sodium restriction reduces cardiac noradrenaline release, but suggest that alterations in sodium intake may influence cardiac noradrenaline metabolism.en
dc.language.isoenen
dc.subject.otherAnimalsen
dc.subject.otherCatecholamines.metabolismen
dc.subject.otherDiet, Sodium-Restricteden
dc.subject.otherDopamine.metabolismen
dc.subject.otherEpinephrine.metabolismen
dc.subject.otherFemaleen
dc.subject.otherGlycols.metabolismen
dc.subject.otherHeart.drug effectsen
dc.subject.otherMethoxyhydroxyphenylglycol.metabolismen
dc.subject.otherMyocardium.metabolismen
dc.subject.otherNorepinephrine.metabolismen
dc.subject.otherOrgan Sizeen
dc.subject.otherRadioimmunoassayen
dc.subject.otherRatsen
dc.subject.otherRats, Inbred Strainsen
dc.titleEffects of sodium restriction on cardiac 3,4-dihydroxyphenylethylene glycol and catecholamine levels in rats.en
dc.typeJournal Articleen
dc.identifier.journaltitleClinical and Experimental Pharmacology & Physiologyen
dc.identifier.affiliationUniversity of Melbourne, Department of Medicine, Austin Hospital, Heidelberg, Victoria, Australiaen
dc.description.pages671-4en
dc.relation.urlhttps://pubmed.ncbi.nlm.nih.gov/2791336en
dc.type.austinJournal Articleen
item.openairetypeJournal Article-
item.fulltextNo Fulltext-
item.languageiso639-1en-
item.openairecristypehttp://purl.org/coar/resource_type/c_18cf-
item.grantfulltextnone-
item.cerifentitytypePublications-
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