Please use this identifier to cite or link to this item: https://ahro.austin.org.au/austinjspui/handle/1/10150
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dc.contributor.authorMooser, Ven
dc.contributor.authorKatopothis, Aen
dc.contributor.authorCasley, David Jen
dc.contributor.authorJohnston, Colin Ien
dc.date.accessioned2015-05-15T23:30:37Z
dc.date.available2015-05-15T23:30:37Z
dc.date.issued1991-12-01en
dc.identifier.citationJournal of Hypertension. Supplement; 9(6): S114-5en
dc.identifier.govdoc1667995en
dc.identifier.otherPUBMEDen
dc.identifier.urihttp://ahro.austin.org.au/austinjspui/handle/1/10150en
dc.language.isoenen
dc.subject.otherAngiotensin II.blooden
dc.subject.otherAnimalsen
dc.subject.otherCardiomegaly.etiology.metabolism.physiopathologyen
dc.subject.otherDesoxycorticosteroneen
dc.subject.otherHypertension.chemically induced.complications.metabolismen
dc.subject.otherHypertension, Renovascular.complications.metabolism.physiopathologyen
dc.subject.otherHypertrophyen
dc.subject.otherKidney.metabolism.pathologyen
dc.subject.otherMyocardium.metabolismen
dc.subject.otherPeptidyl-Dipeptidase A.metabolismen
dc.subject.otherRatsen
dc.subject.otherRats, Inbred Strainsen
dc.subject.otherSodium Chlorideen
dc.titleCardiac and renal hypertrophy is independent of tissue angiotensin converting enzyme and circulating angiotensin II in hypertensive rats.en
dc.typeJournal Articleen
dc.identifier.journaltitleJournal of Hypertension. Supplementen
dc.identifier.affiliationDepartment of Medicine, University of Melbourne, Austin Hospital, Heidelberg, Victoria, Australiaen
dc.description.pagesS114-5en
dc.relation.urlhttps://pubmed.ncbi.nlm.nih.gov/1667995en
dc.type.austinJournal Articleen
item.languageiso639-1en-
item.openairecristypehttp://purl.org/coar/resource_type/c_18cf-
item.openairetypeJournal Article-
item.grantfulltextnone-
item.cerifentitytypePublications-
item.fulltextNo Fulltext-
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