Please use this identifier to cite or link to this item: https://ahro.austin.org.au/austinjspui/handle/1/10046
Title: Clinical modifiers for heart failure following myocardial infarction.
Austin Authors: Anavekar, Nandan S;Anavekar, Nagesh S
Affiliation: Clinical Pharmacology & Therapeutics, University of Melbourne, Austin Health, Studley Road, Heidelberg 3084, Australia
Issue Date: 1-Dec-2005
Publication information: Current Heart Failure Reports; 2(4): 165-73
Abstract: Heart failure (HF) is a clinical syndrome that occurs when the ability of the heart to meet the requirements of the body fails. Myocardial infarction (MI) is a common antecedent event that predisposes a patient to HF. Loss of cardiac function following MI occurs in the context of myocyte death and ventricular remodeling. The clinical significance of HF following MI is underscored by the fact that among MI survivors, the risk of death is markedly elevated in those who develop HF compared with those who do not. Various modifying factors associated with the development of HF following MI have been identified. Use of multimodality therapy with improved clinical outcomes for HF has increased the need to specifically identify the failing heart at an earlier stage. The ability to identify heart failure early in its pathogenesis will enable finer risk stratification following MI. This article reviews various risk predictors for the development of HF following MI.
Gov't Doc #: 16332309
URI: http://ahro.austin.org.au/austinjspui/handle/1/10046
URL: https://pubmed.ncbi.nlm.nih.gov/16332309
Type: Journal Article
Subjects: Biological Markers.blood
Cytokines.blood
Echocardiography
Exercise Test
Heart Failure.blood.diagnosis.etiology
Humans
Myocardial Infarction.complications
Natriuretic Peptide, Brain.blood
Prognosis
Risk Factors
Stroke Volume.physiology
Troponin.blood
Appears in Collections:Journal articles

Show full item record

Page view(s)

6
checked on Dec 2, 2022

Google ScholarTM

Check


Items in AHRO are protected by copyright, with all rights reserved, unless otherwise indicated.