Please use this identifier to cite or link to this item: https://ahro.austin.org.au/austinjspui/handle/1/22960
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dc.contributor.authorDawson, Luke P-
dc.contributor.authorCole, Justin A-
dc.contributor.authorLancefield, Terase F-
dc.contributor.authorAjani, Andrew E-
dc.contributor.authorAndrianopoulos, Nick-
dc.contributor.authorThrift, Amanda G-
dc.contributor.authorClark, David J-
dc.contributor.authorBrennan, Angela L-
dc.contributor.authorFreeman, Melanie-
dc.contributor.authorO'Brien, Jessica-
dc.contributor.authorSebastian, Martin-
dc.contributor.authorChan, William-
dc.contributor.authorShaw, James A-
dc.contributor.authorDinh, Diem-
dc.contributor.authorReid, Christopher M-
dc.contributor.authorDuffy, Stephen J-
dc.date2020-04-04-
dc.date.accessioned2020-04-14T04:01:24Z-
dc.date.available2020-04-14T04:01:24Z-
dc.date.issued2020-10-
dc.identifier.citationInternational Journal of Stroke 2020; 15(8): 909-922en_US
dc.identifier.urihttps://ahro.austin.org.au/austinjspui/handle/1/22960-
dc.description.abstractStroke rates and risk factors may change as percutaneous coronary intervention practice evolves and no data are available comparing stroke incidence after percutaneous coronary intervention to the general population. This study aimed to identify the incidence and risk factors for inpatient and subsequent stroke following percutaneous coronary intervention with comparison to age-matched controls. Data were prospectively collected from 22,618 patients undergoing percutaneous coronary intervention in the Melbourne Interventional Group registry (2005-2015). The cohort was compared to the North-East Melbourne Stroke Incidence Study population-based cohort (1997-1999) and predefined variables assessed for association with inpatient or outpatient stroke. Inpatient stroke occurred in 0.33% (65.3% ischemic, 28.0% haemorrhagic, and 6.7% cause unknown), while outpatient stroke occurred in 0.55%. Inpatient and outpatient stroke were associated with higher rates of in-hospital major adverse cardiovascular outcomes (p < 0.0001) and mortality (p < 0.0001), as well as 12-month mortality (p < 0.0001). Factors independently associated with inpatient stroke were renal impairment, ST-elevation myocardial infarction, previous stroke, left ventricular ejection fraction 30-45%, and female sex, while those associated with outpatient stroke were previous stroke, chronic lung disease, previous myocardial infarction, rheumatoid arthritis, female sex, and older age. Compared to the age-standardized population-based cohort, stroke rates in the 12 months following discharge were higher for percutaneous coronary intervention patients <65 years old, but lower for percutaneous coronary intervention patients ≥65 years old. Risk of inpatient stroke following percutaneous coronary intervention appears to be largely associated with clinical status at presentation, while outpatient stroke relates more to age and chronic disease. Compared to the general population, outpatient stroke rates following percutaneous coronary intervention are higher for younger, but not older, patients.en_US
dc.language.isoeng-
dc.subjectStrokeen_US
dc.subjectclinical outcomesen_US
dc.subjectpercutaneous coronary interventionen_US
dc.subjectrisk factorsen_US
dc.titleIncidence and risk factors for stroke following percutaneous coronary intervention.en_US
dc.typeJournal Articleen_US
dc.identifier.journaltitleInternational Journal of Strokeen_US
dc.identifier.affiliationStroke and Ageing Research, Department of Medicine, School of Clinical Sciences at Monash Health, Monash University, Melbourne, Australiaen_US
dc.identifier.affiliationDepartment of Epidemiology and Preventive Medicine, Monash University, Melbourne, Australiaen_US
dc.identifier.affiliationSchool of Public Health, Curtin University, Perth, Australiaen_US
dc.identifier.affiliationDepartment of Cardiology, University Hospital Geelong, Geelong, Australiaen_US
dc.identifier.affiliationCardiologyen_US
dc.identifier.affiliationDepartment of Cardiology, Box Hill Hospital, Melbourne, Australiaen_US
dc.identifier.affiliationDepartment of Cardiovascular Medicine, The Alfred Hospital, Melbourne, Australiaen_US
dc.identifier.affiliationDepartment of Cardiology, Royal Melbourne Hospital, Melbourne, Australiaen_US
dc.identifier.affiliationBaker IDI Heart and Diabetes Institute, Melbourne, Australiaen_US
dc.identifier.doi10.1177/1747493020912607en_US
dc.type.contentTexten_US
dc.identifier.orcid0000-0003-3789-5808en_US
dc.identifier.orcid0000-0001-8533-4170en_US
dc.identifier.pubmedid32248767-
dc.type.austinJournal Article-
local.name.researcherClark, David J
item.openairecristypehttp://purl.org/coar/resource_type/c_18cf-
item.cerifentitytypePublications-
item.fulltextNo Fulltext-
item.grantfulltextnone-
item.languageiso639-1en-
item.openairetypeJournal Article-
crisitem.author.deptCardiology-
crisitem.author.deptUniversity of Melbourne Clinical School-
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