Please use this identifier to cite or link to this item: http://ahro.austin.org.au/austinjspui/handle/1/13635
Title: Pathophysiology of diabetic nephropathy.
Authors: Cooper, Mark E;Gilbert, Richard E;Epstein, M
Affiliation: Department of Medicine, University of Melbourne, Austin & Repatriation Medical Centre, Heidelberg, Victoria, Australia.
Issue Date: 1-Dec-1998
Citation: Metabolism: Clinical and Experimental; 47(12 Suppl 1): 3-6
Abstract: Diabetic nephropathy (DN) is now the commonest cause of end-stage renal failure in the Western world. Recent studies examining the pathogenesis of diabetic complications have focused on the complex interaction between genetic and hemodynamic mechanisms in addition to metabolic factors such as advanced glycation, protein kinase C (PKC) activation, and polyol production. The importance of the various components, particularly with regard to the progression of DN, is currently being explored with the assistance of targeted drug intervention studies.
Internal ID Number: 9867062
URI: http://ahro.austin.org.au/austinjspui/handle/1/13635
URL: http://www.ncbi.nlm.nih.gov/pubmed/9867062
Type: Journal Article
Subjects: Diabetic Nephropathies.genetics.metabolism.physiopathology.therapy
Glycosylation End Products, Advanced.metabolism
Hemodynamics.physiology
Humans
Polymers.metabolism
Appears in Collections:Journal articles

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